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Aspirin inhibits LPS-induced macrophage activation via the NF-κB pathway |  Scientific Reports
Aspirin inhibits LPS-induced macrophage activation via the NF-κB pathway | Scientific Reports

Anti-inflammatory effect of hispidin on LPS induced macrophage inflammation  through MAPK and JAK1/STAT3 signaling pathways | Applied Biological  Chemistry | Full Text
Anti-inflammatory effect of hispidin on LPS induced macrophage inflammation through MAPK and JAK1/STAT3 signaling pathways | Applied Biological Chemistry | Full Text

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports

SRV2 promotes mitochondrial fission and Mst1-Drp1 signaling in LPS-induced  septic cardiomyopathy | Aging
SRV2 promotes mitochondrial fission and Mst1-Drp1 signaling in LPS-induced septic cardiomyopathy | Aging

Dexmedetomidine inhibits LPS-induced proinflammatory responses via  suppressing HIF1α-dependent glycolysis in macrophages | Aging
Dexmedetomidine inhibits LPS-induced proinflammatory responses via suppressing HIF1α-dependent glycolysis in macrophages | Aging

MAR1 suppresses inflammatory response in LPS-induced RAW 264.7 macrophages  and human primary peripheral blood mononuclear cells via the  SIRT1/PGC-1α/PPAR-γ pathway | Journal of Inflammation | Full Text
MAR1 suppresses inflammatory response in LPS-induced RAW 264.7 macrophages and human primary peripheral blood mononuclear cells via the SIRT1/PGC-1α/PPAR-γ pathway | Journal of Inflammation | Full Text

Lipopolysaccharide-Induced Microglial Activation and Neuroprotection  against Experimental Brain Injury Is Independent of Hematogenous TLR4 |  Journal of Neuroscience
Lipopolysaccharide-Induced Microglial Activation and Neuroprotection against Experimental Brain Injury Is Independent of Hematogenous TLR4 | Journal of Neuroscience

LPS‑induced proinflammatory cytokine expression in human airway epithelial  cells and macrophages via NF‑κB, STAT3 or AP‑1 activation
LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation

AP pre-treatment repressed LPS-induced overproduction of inflammatory... |  Download Scientific Diagram
AP pre-treatment repressed LPS-induced overproduction of inflammatory... | Download Scientific Diagram

Potentiation of LPS-Induced Apoptotic Cell Death in Human Hepatoma HepG2  Cells by Aspirin via ROS and Mitochondrial Dysfunction: Protection by  N-Acetyl Cysteine | PLOS ONE
Potentiation of LPS-Induced Apoptotic Cell Death in Human Hepatoma HepG2 Cells by Aspirin via ROS and Mitochondrial Dysfunction: Protection by N-Acetyl Cysteine | PLOS ONE

Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and  neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice -  ScienceDirect
Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice - ScienceDirect

MiR-200c-3p inhibits LPS-induced M1 polarization of BV2 cells by targeting  RIP2 | SpringerLink
MiR-200c-3p inhibits LPS-induced M1 polarization of BV2 cells by targeting RIP2 | SpringerLink

Effect of LPS-treatment on pro-inflammatory cytokine/chemokine levels.... |  Download Scientific Diagram
Effect of LPS-treatment on pro-inflammatory cytokine/chemokine levels.... | Download Scientific Diagram

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports

Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an  Apolipoprotein AI Mimetic Peptide | Circulation Research
Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an Apolipoprotein AI Mimetic Peptide | Circulation Research

Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory  Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts
Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts

Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through  Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway |  ACS Omega
Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway | ACS Omega

Effect of LPS treatment of RB family protein expression. Western... |  Download Scientific Diagram
Effect of LPS treatment of RB family protein expression. Western... | Download Scientific Diagram

LPS‑induced proinflammatory cytokine expression in human airway epithelial  cells and macrophages via NF‑κB, STAT3 or AP‑1 activation
LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation

A unique hybrid characteristic having both pro- and anti-inflammatory  phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4  microglia | Scientific Reports
A unique hybrid characteristic having both pro- and anti-inflammatory phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4 microglia | Scientific Reports

Lipopolysaccharide (LPS)-Induced Biliary Epithelial Cell NRas Activation  Requires Epidermal Growth Factor Receptor (EGFR) | PLOS ONE
Lipopolysaccharide (LPS)-Induced Biliary Epithelial Cell NRas Activation Requires Epidermal Growth Factor Receptor (EGFR) | PLOS ONE

Dynamin‐related Irgm proteins modulate LPS‐induced caspase‐11 activation  and septic shock | EMBO reports
Dynamin‐related Irgm proteins modulate LPS‐induced caspase‐11 activation and septic shock | EMBO reports

Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in  Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 |  Infection and Immunity
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity

Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by  Internalization in Teleost Macrophages
Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by Internalization in Teleost Macrophages

Effect of LPS treatment on cell number, DNA fragmentation, and... |  Download Scientific Diagram
Effect of LPS treatment on cell number, DNA fragmentation, and... | Download Scientific Diagram

The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus  Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell
The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell

Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in  Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 |  Infection and Immunity
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity

LPS Model of Sepsis | Melior Discovery
LPS Model of Sepsis | Melior Discovery

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports